PhD, University of Florida
cristina.colomerwinter [at] unige.ch
Cristina is originally from Barcelona, Spain. She graduated from the Autonomous University of Barcelona with a bachelor’s degree in Biology and completed during this time, several internships in Germany. After receiving her master’s degree in Microbiology at the University of Barcelona, Cristina worked as a R&D scientist at the pharmaceutical company B.Braun developing antibacterial medical devices.
In 2013, Cristina moved to the University of Rochester (New York) where she joined the group of Dr. Jose Lemos to begin her dissertation work on the pathophysiology of Enterococcus faecalis. After transferring to the University of Florida (Florida), she continued her work under the mentorship of Dr. Lemos. Her PhD work investigated the role of the global stress response regulator (p)ppGpp and of manganese transport to the systemic virulence of E. faecalis. In 2016 she received an American Heart Association Predoctoral Fellowship for her work.
After successfully completing her doctoral degree in 2018, Cristina moved back to Barcelona and worked as a QA Scientist for the pharmaceutical company Boehringer-Ingelheim. In 2022, she joined the Kline lab at the University of Geneva to work on enterococcal adaptation to the host environment during infection. When not in the lab, you will most likely find Cristina enjoying her time off, be it at the gym, in a restaurant, on a mountain, or traveling around the world.
|1.||Survival of the fittest: the relationship of (p)ppGpp with bacterial virulence. Kundra S, Colomer-Winter C, Lemos JA; Front Microbiol. 2020 (Review)|
|2.||(p)ppGpp and CodY promote Enterococcus faecalis virulence in a murine model of catheter-associated urinary tract infection. Colomer-Winter C*, Flores-Mireles AL*, Kundra S, Hultgren SJ, Lemos JA; mSphere 2019.|
|3.||Biofilm assays on fibrinogen-coated silicone catheters and 96-well polystyrene plates. Colomer-Winter C, Lemos JA, Flores-Mireles AL; Bio Protoc. 2019|
|4.||Cellular levels of (p)ppGpp differentially affect the pathogenesis of infective endocarditis in Enterococcus faecalis. Colomer-Winter C, Gaca AO, Chuang-Smith ON, Lemos JA, Frank KL; Microbiology 2018|
|5.||Manganese acquisition is essential for virulence of Enterococcus faecalis. Colomer-Winter C, Flores-Mireles AL, Baker SP, Frank KL, Lynch AJL, Hultgren SJ, Kitten T, Lemos JA; PLoS Pathog. 2018|
|6.||Association of metal homeostasis and (p)ppGpp regulation in the pathophysiology of Enterococcus faecalis. Colomer-Winter C, Gaca AO, Lemos JA; Infect. Immun. 2017|
|7.||From (p)ppGpp to (pp)pGpp: Characterization of regulatory effects of pGpp synthesized by the Small Alarmone Synthetase of Enterococcus faecalis. Gaca AO, Kudrin P, Colomer-Winter C, Beljantseva J, Liu K, Anderson B, Wang JD, Rejman D, Potrykus K, Cashel M, Hauryliuk V, Lemos JA; J. Bacteriol. 2015|
|8.||Many means to a common end: the intricacies of (p)ppGpp metabolism and its control of bacterial homeostasis. Gaca AO, Colomer-Winter C, Lemos JA. J. Bacteriol. 2015 (Review)|
|9.||Transcriptome analysis of Enterococcus faecalis during mammalian infection shows cells undergo adaptation and exist in a stringent response state. Frank KL, Colomer-Winter C, Grindle SM, Lemos JA, Schlievert PM, Dunny GM; PLoS One 2014.|